Take DAT, Flu!

Lior Lobel; Wendy S. Garrett

doi:https://doi.org/10.1016/j.immuni.2017.09.002

Take DAT, Flu!

Lior Lobel, Wendy S. Garrett

Research output: Contribution to journal › Short survey › peer-review

Abstract

Some microbial metabolites can be immunomodulatory, but there is limited understanding of how these contribute to inter-individual variation in response to infection. In a recent study in Science, Steed et al. (2017) show that the bacterial metabolite desaminotyrosine (DAT) increases type I interferon expression, resulting in an improved immune response to influenza infection. Some microbial metabolites can be immunomodulatory, but there is limited understanding of how these contribute to inter-individual variation in response to infection. In a recent study in Science, Steed et al. (2017) show that the bacterial metabolite desaminotyrosine (DAT) increases type I interferon expression, resulting in an improved immune response to influenza infection.

Original language	English
Pages (from-to)	400-402
Number of pages	3
Journal	Immunity
Volume	47
Issue number	3
DOIs	https://doi.org/10.1016/j.immuni.2017.09.002
State	Published - 19 Sep 2017
Externally published	Yes

All Science Journal Classification (ASJC) codes

Infectious Diseases
Immunology and Allergy
Immunology

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

Access to Document

https://doi.org/10.1016/j.immuni.2017.09.002

Cite this

@article{610ea126f2ac4b0eae0131cd7f7a8cc4,

title = "Take DAT, Flu!",

abstract = "Some microbial metabolites can be immunomodulatory, but there is limited understanding of how these contribute to inter-individual variation in response to infection. In a recent study in Science, Steed et al. (2017) show that the bacterial metabolite desaminotyrosine (DAT) increases type I interferon expression, resulting in an improved immune response to influenza infection. Some microbial metabolites can be immunomodulatory, but there is limited understanding of how these contribute to inter-individual variation in response to infection. In a recent study in Science, Steed et al. (2017) show that the bacterial metabolite desaminotyrosine (DAT) increases type I interferon expression, resulting in an improved immune response to influenza infection.",

author = "Lior Lobel and Garrett, {Wendy S.}",

note = "Publisher Copyright: {\textcopyright} 2017 Elsevier Inc.",

year = "2017",

month = sep,

day = "19",

doi = "https://doi.org/10.1016/j.immuni.2017.09.002",

language = "الإنجليزيّة",

volume = "47",

pages = "400--402",

journal = "Immunity",

issn = "1074-7613",

publisher = "Cell Press",

number = "3",

}

TY - JOUR

T1 - Take DAT, Flu!

AU - Lobel, Lior

AU - Garrett, Wendy S.

PY - 2017/9/19

Y1 - 2017/9/19

N2 - Some microbial metabolites can be immunomodulatory, but there is limited understanding of how these contribute to inter-individual variation in response to infection. In a recent study in Science, Steed et al. (2017) show that the bacterial metabolite desaminotyrosine (DAT) increases type I interferon expression, resulting in an improved immune response to influenza infection. Some microbial metabolites can be immunomodulatory, but there is limited understanding of how these contribute to inter-individual variation in response to infection. In a recent study in Science, Steed et al. (2017) show that the bacterial metabolite desaminotyrosine (DAT) increases type I interferon expression, resulting in an improved immune response to influenza infection.

AB - Some microbial metabolites can be immunomodulatory, but there is limited understanding of how these contribute to inter-individual variation in response to infection. In a recent study in Science, Steed et al. (2017) show that the bacterial metabolite desaminotyrosine (DAT) increases type I interferon expression, resulting in an improved immune response to influenza infection. Some microbial metabolites can be immunomodulatory, but there is limited understanding of how these contribute to inter-individual variation in response to infection. In a recent study in Science, Steed et al. (2017) show that the bacterial metabolite desaminotyrosine (DAT) increases type I interferon expression, resulting in an improved immune response to influenza infection.

UR - http://www.scopus.com/inward/record.url?scp=85031756832&partnerID=8YFLogxK

U2 - https://doi.org/10.1016/j.immuni.2017.09.002

DO - https://doi.org/10.1016/j.immuni.2017.09.002

M3 - عرض مختصر

C2 - 28930655

SN - 1074-7613

VL - 47

SP - 400

EP - 402

JO - Immunity

JF - Immunity

IS - 3

ER -

Take DAT, Flu!

Abstract

All Science Journal Classification (ASJC) codes

UN SDGs

Access to Document

Other files and links

Fingerprint

Cite this