Rac1 functions downstream of miR-142 in regulation of erythropoiesis

Natalia Rivkin, Elik Chapnik, Yehudit Birger, Eran Yanowski, Caterina Curato, Alexander Mildner, Ziv Porat, Gail Amir, Shai Izraeli, Steffen Jung, Eran Hornstein

Research output: Contribution to journalLetterpeer-review

Abstract

Hematopoietic-specific miR-142 is a critical regulator of various blood cell lineages including CD4+ dendritic cells1 and platelet biogenesis in megakaryocytes.2 Furthermore, we recently reported that miR-142 is required in order to maintain the biconcave shape of erythrocytes, their structural resilience and lifespan, through a mechanism that involves actin filament homeostasis.3 Here, we used a mouse loss of function allele to characterize a new axis, where miR-142 functions upstream of Rac1 in regulating erythropoiesis.
Original languageEnglish
Pages (from-to)476-480
Number of pages5
JournalHaematologica
Volume102
Issue number12
DOIs
StatePublished - 30 Nov 2017

All Science Journal Classification (ASJC) codes

  • Hematology

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