NF-κB Regulation by NLRs: T Cells Join the Club

Christoph A. Thaiss; Eran Elinav

doi:10.1016/j.immuni.2015.03.010

NF-κB Regulation by NLRs: T Cells Join the Club

Christoph A. Thaiss
, Eran Elinav

Department of Systems Immunology

Weizmann Institute of Science

Research output: Contribution to journal › Short survey › peer-review

Abstract

NLRP12 is an innate immune receptor whose regulation of NF-κB signaling in myeloid cells is critical in preventing multiple auto-inflammatory diseases. In this issue of Immunity, Lukens etal. (2015) show that a similar NLRP12-mediated mechanism is functionally important in Tcells.

Original language	English
Pages (from-to)	595-597
Number of pages	3
Journal	Immunity
Volume	42
Issue number	4
DOIs	https://doi.org/10.1016/j.immuni.2015.03.010
State	Published - 21 Apr 2015

ASJC Scopus subject areas

Immunology and Allergy
Immunology
Infectious Diseases

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10.1016/j.immuni.2015.03.010

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@article{e00cf69f4fba4dc8a06e022005769932,

title = "NF-κB Regulation by NLRs: T Cells Join the Club",

abstract = "NLRP12 is an innate immune receptor whose regulation of NF-κB signaling in myeloid cells is critical in preventing multiple auto-inflammatory diseases. In this issue of Immunity, Lukens etal. (2015) show that a similar NLRP12-mediated mechanism is functionally important in Tcells.",

author = "Thaiss, \{Christoph A.\} and Eran Elinav",

note = "Publisher Copyright: {\textcopyright} 2015 Elsevier Inc.",

year = "2015",

month = apr,

day = "21",

doi = "10.1016/j.immuni.2015.03.010",

language = "English",

volume = "42",

pages = "595--597",

journal = "Immunity",

issn = "1074-7613",

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number = "4",

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TY - JOUR

T1 - NF-κB Regulation by NLRs

T2 - T Cells Join the Club

AU - Thaiss, Christoph A.

AU - Elinav, Eran

PY - 2015/4/21

Y1 - 2015/4/21

N2 - NLRP12 is an innate immune receptor whose regulation of NF-κB signaling in myeloid cells is critical in preventing multiple auto-inflammatory diseases. In this issue of Immunity, Lukens etal. (2015) show that a similar NLRP12-mediated mechanism is functionally important in Tcells.

AB - NLRP12 is an innate immune receptor whose regulation of NF-κB signaling in myeloid cells is critical in preventing multiple auto-inflammatory diseases. In this issue of Immunity, Lukens etal. (2015) show that a similar NLRP12-mediated mechanism is functionally important in Tcells.

UR - https://www.scopus.com/pages/publications/84928180625

U2 - 10.1016/j.immuni.2015.03.010

DO - 10.1016/j.immuni.2015.03.010

M3 - Short survey

SN - 1074-7613

VL - 42

SP - 595

EP - 597

JO - Immunity

JF - Immunity

IS - 4

ER -

NF-κB Regulation by NLRs: T Cells Join the Club

Abstract

ASJC Scopus subject areas

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