NF-κB Regulation by NLRs: T Cells Join the Club

Christoph A. Thaiss; Eran Elinav

doi:10.1016/j.immuni.2015.03.010

NF-κB Regulation by NLRs: T Cells Join the Club

Christoph A. Thaiss, Eran Elinav

Department of Systems Immunology

Weizmann Institute of Science

Research output: Contribution to journal › Short survey › peer-review

Abstract

NLRP12 is an innate immune receptor whose regulation of NF-κB signaling in myeloid cells is critical in preventing multiple auto-inflammatory diseases. In this issue of Immunity, Lukens etal. (2015) show that a similar NLRP12-mediated mechanism is functionally important in Tcells.

Original language	English
Pages (from-to)	595-597
Number of pages	3
Journal	Immunity
Volume	42
Issue number	4
DOIs	https://doi.org/10.1016/j.immuni.2015.03.010
State	Published - 21 Apr 2015

All Science Journal Classification (ASJC) codes

Infectious Diseases
Immunology and Allergy
Immunology

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10.1016/j.immuni.2015.03.010

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title = "NF-κB Regulation by NLRs: T Cells Join the Club",

abstract = "NLRP12 is an innate immune receptor whose regulation of NF-κB signaling in myeloid cells is critical in preventing multiple auto-inflammatory diseases. In this issue of Immunity, Lukens etal. (2015) show that a similar NLRP12-mediated mechanism is functionally important in Tcells.",

author = "Thaiss, {Christoph A.} and Eran Elinav",

note = "Publisher Copyright: {\textcopyright} 2015 Elsevier Inc.",

year = "2015",

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doi = "10.1016/j.immuni.2015.03.010",

language = "الإنجليزيّة",

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AU - Thaiss, Christoph A.

AU - Elinav, Eran

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N2 - NLRP12 is an innate immune receptor whose regulation of NF-κB signaling in myeloid cells is critical in preventing multiple auto-inflammatory diseases. In this issue of Immunity, Lukens etal. (2015) show that a similar NLRP12-mediated mechanism is functionally important in Tcells.

AB - NLRP12 is an innate immune receptor whose regulation of NF-κB signaling in myeloid cells is critical in preventing multiple auto-inflammatory diseases. In this issue of Immunity, Lukens etal. (2015) show that a similar NLRP12-mediated mechanism is functionally important in Tcells.

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U2 - 10.1016/j.immuni.2015.03.010

DO - 10.1016/j.immuni.2015.03.010

M3 - عرض مختصر

SN - 1074-7613

VL - 42

SP - 595

EP - 597

JO - Immunity

JF - Immunity

IS - 4

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NF-κB Regulation by NLRs: T Cells Join the Club

Abstract

All Science Journal Classification (ASJC) codes

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