Integrin Alpha E (CD103) Limits Virus-Induced IFN-I Production in Conventional Dendritic Cells

Vikas Duhan, Vishal Khairnar, Simo Kitanovski, Thamer A Hamdan, Andrés D Klein, Judith Lang, Murtaza Ali, Tom Adomati, Hilal Bhat, Sarah-Kim Friedrich, Fanghui Li, Philippe Krebs, Anthony H Futerman, Marylyn M Addo, Cornelia Hardt, Daniel Hoffmann, Philipp A Lang, Karl S Lang

Research output: Contribution to journalArticlepeer-review

Abstract

Early and strong production of IFN-I by dendritic cells is important to control vesicular stomatitis virus (VSV), however mechanisms which explain this cell-type specific innate immune activation remain to be defined. Here, using a genome wide association study (GWAS), we identified Integrin alpha-E (Itgae, CD103) as a new regulator of antiviral IFN-I production in a mouse model of vesicular stomatitis virus (VSV) infection. CD103 was specifically expressed by splenic conventional dendritic cells (cDCs) and limited IFN-I production in these cells during VSV infection. Mechanistically, CD103 suppressed AKT phosphorylation and mTOR activation in DCs. Deficiency in CD103 accelerated early IFN-I in cDCs and prevented death in VSV infected animals. In conclusion, CD103 participates in regulation of cDC specific IFN-I induction and thereby influences immune activation after VSV infection.

Original languageEnglish
Article number607889
Number of pages11
JournalFrontiers in Immunology
Volume11
DOIs
StatePublished - 27 Jan 2021

All Science Journal Classification (ASJC) codes

  • Immunology and Allergy
  • Immunology

Fingerprint

Dive into the research topics of 'Integrin Alpha E (CD103) Limits Virus-Induced IFN-I Production in Conventional Dendritic Cells'. Together they form a unique fingerprint.

Cite this