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ICAMs Are Not Obligatory for Functional Immune Synapses between Naive CD4 T Cells and Lymph Node DCs

  • Sara W. Feigelson
  • , Adam Solomon
  • , Adi Biram
  • , Miki Hatzav
  • , Moria Lichtenstein
  • , Ofer Regev
  • , Stav Kozlovski
  • , Diana Varol
  • , Caterina Curato
  • , Dena Leshkowitz
  • , Steffen Jung
  • , Ziv Shulman
  • , Ronen Alon

Research output: Contribution to journalArticlepeer-review

Abstract

Protective immune responses depend on the formation of immune synapses between T cells and antigen-presenting cells (APCs). The two main LFA-1 ligands, ICAM-1 and ICAM-2, are co-expressed on many cell types, including APCs and blood vessels. Although these molecules were suggested to be key players in immune synapses studied in vitro, their contribution to helper T cell priming in vivo is unclear. Here, we used transgenic mice and intravital imaging to examine the role of dendritic cell (DC) ICAM-1 and ICAM-2 in naive CD4 T cell priming and differentiation in skin-draining lymph nodes. Surprisingly, ICAM deficiency on endogenous CD40-stimulated lymph node DCs did not impair their ability to arrest and prime CD4 lymphocyte activation and differentiation into Th1 and Tfh effectors. Thus, functional T cell receptor (TCR)-specific helper T cell synapses with antigen-presenting DCs and subsequent proliferation and early differentiation into T effectors do not require LFA-1-mediated T cell adhesiveness to DC ICAMs.

Original languageEnglish GB
Pages (from-to)849-859
Number of pages11
JournalCell Reports
Volume22
Issue number4
DOIs
StatePublished - 23 Jan 2018

ASJC Scopus subject areas

  • General Biochemistry,Genetics and Molecular Biology

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