ATG9 raises the BAR for PI4P in autophagy

Oren Shatz; Zvulun Elazar

doi:10.1083/jcb.201904063

ATG9 raises the BAR for PI4P in autophagy

Oren Shatz, Zvulun Elazar

Department of Biomolecular Sciences

Weizmann Institute of Science

Research output: Contribution to journal › Article › peer-review

Abstract

ATG9 vesicles are crucial for autophagy, yet the role of ATG9 remains unclear. In this issue, Judith et al. (2019. J. Cell Biol. https://doi.org/10.1083/jcb.201901115) implicate the BAR protein Arfaptin2 in the loading of PI4-kinase IIIβ onto ATG9 vesicles for recruitment of ATG13 to the site of autophagosome biogenesis.

Original language	English
Pages (from-to)	1432-1433
Number of pages	2
Journal	Journal of Cell Biology
Volume	218
Issue number	5
DOIs	https://doi.org/10.1083/jcb.201904063
State	Published - 19 Apr 2019

All Science Journal Classification (ASJC) codes

Cell Biology

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title = "ATG9 raises the BAR for PI4P in autophagy",

abstract = "ATG9 vesicles are crucial for autophagy, yet the role of ATG9 remains unclear. In this issue, Judith et al. (2019. J. Cell Biol. https://doi.org/10.1083/jcb.201901115) implicate the BAR protein Arfaptin2 in the loading of PI4-kinase IIIβ onto ATG9 vesicles for recruitment of ATG13 to the site of autophagosome biogenesis.",

author = "Oren Shatz and Zvulun Elazar",

note = "We apologize to the authors whose work was not cited here due to citation restrictions. Z. Elazar is an incumbent of the Harold Korda Chair of Biology. We are grateful for funding from the Israel Science Foundation (grant 1247/15), the Legacy Heritage Fund (grant 1935/16), and the Yeda-Sela Center for Basic Research. The authors declare no competing financial interests. Funding: Israel Science Foundation (1247/15) Legacy Heritage Fund (1935/16)",

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doi = "10.1083/jcb.201904063",

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T1 - ATG9 raises the BAR for PI4P in autophagy

AU - Shatz, Oren

AU - Elazar, Zvulun

N1 - We apologize to the authors whose work was not cited here due to citation restrictions. Z. Elazar is an incumbent of the Harold Korda Chair of Biology. We are grateful for funding from the Israel Science Foundation (grant 1247/15), the Legacy Heritage Fund (grant 1935/16), and the Yeda-Sela Center for Basic Research. The authors declare no competing financial interests. Funding: Israel Science Foundation (1247/15) Legacy Heritage Fund (1935/16)

PY - 2019/4/19

Y1 - 2019/4/19

N2 - ATG9 vesicles are crucial for autophagy, yet the role of ATG9 remains unclear. In this issue, Judith et al. (2019. J. Cell Biol. https://doi.org/10.1083/jcb.201901115) implicate the BAR protein Arfaptin2 in the loading of PI4-kinase IIIβ onto ATG9 vesicles for recruitment of ATG13 to the site of autophagosome biogenesis.

AB - ATG9 vesicles are crucial for autophagy, yet the role of ATG9 remains unclear. In this issue, Judith et al. (2019. J. Cell Biol. https://doi.org/10.1083/jcb.201901115) implicate the BAR protein Arfaptin2 in the loading of PI4-kinase IIIβ onto ATG9 vesicles for recruitment of ATG13 to the site of autophagosome biogenesis.

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U2 - 10.1083/jcb.201904063

DO - 10.1083/jcb.201904063

M3 - مقالة

C2 - 31003996

SN - 0021-9525

VL - 218

SP - 1432

EP - 1433

JO - Journal of Cell Biology

JF - Journal of Cell Biology

IS - 5

ER -

ATG9 raises the BAR for PI4P in autophagy

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