A TRIM16-Galactin3 Complex Mediates Autophagy of Damaged Endomembranes

Research output: Contribution to journalEditorialpeer-review


Selective autophagy, an essential process for maintaining intracellular homeostasis, depends on precise target recognition and local activation. Reporting in Developmental Cell, Chauhan et al. (2016) elegantly demonstrate that interaction between TRIM16 and Galectin3 orchestrates the recruitment of core autophagic factors and activates selective autophagy at the site of damaged endomembranes.
Original languageEnglish
Pages (from-to)1-2
Number of pages2
JournalDevelopmental Cell
Issue number1
StatePublished - 10 Oct 2016


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