A TRIM16-Galactin3 Complex Mediates Autophagy of Damaged Endomembranes

Milana Fraiberg, Zvulun Elazar

Research output: Contribution to journalShort surveypeer-review

Abstract

Selective autophagy, an essential process for maintaining intracellular homeostasis, depends on precise target recognition and local activation. Reporting in Developmental Cell, Chauhan et al. (2016) elegantly demonstrate that interaction between TRIM16 and Galectin3 orchestrates the recruitment of core autophagic factors and activates selective autophagy at the site of damaged endomembranes.

Original languageEnglish
Pages (from-to)1-2
Number of pages2
JournalDevelopmental Cell
Volume39
Issue number1
DOIs
StatePublished - 10 Oct 2016

All Science Journal Classification (ASJC) codes

  • General Biochemistry,Genetics and Molecular Biology
  • Molecular Biology
  • Cell Biology
  • Developmental Biology

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